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Corrigendum to "Intradiencephalon injection of histamine inhibited the recovery of locomotor function of spinal cord injured zebrafish" [Biochem. Biophys. Res. Comm. 489 (1) (2017) 275-280].

Biochemical and biophysical research communications

Huang SB, Zhao HD, Wang LF, Sun MF, Zhu YL, Wu YB, Xu YD, Peng SX, Cui C, Shen YQ.
PMID: 28864060
Biochem Biophys Res Commun. 2017 Oct 21;492(3):528. doi: 10.1016/j.bbrc.2017.08.098. Epub 2017 Aug 31.

No abstract available.

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Huang SB, Zhao HD, Wang LF, et al. Corrigendum to "Intradiencephalon injection of histamine inhibited the recovery of locomotor function of spinal cord injured zebrafish" [Biochem. Biophys. Res. Comm. 489 (1) (2017) 275-280]. Biochem Biophys Res Commun. 2017;492(3):528doi: 10.1016/j.bbrc.2017.08.098.
Huang, S. B., Zhao, H. D., Wang, L. F., Sun, M. F., Zhu, Y. L., Wu, Y. B., Xu, Y. D., Peng, S. X., Cui, C., & Shen, Y. Q. (2017). Corrigendum to "Intradiencephalon injection of histamine inhibited the recovery of locomotor function of spinal cord injured zebrafish" [Biochem. Biophys. Res. Comm. 489 (1) (2017) 275-280]. Biochemical and biophysical research communications, 492(3), 528. https://doi.org/10.1016/j.bbrc.2017.08.098
Huang, Shu-Bing, et al. "Corrigendum to "Intradiencephalon injection of histamine inhibited the recovery of locomotor function of spinal cord injured zebrafish" [Biochem. Biophys. Res. Comm. 489 (1) (2017) 275-280]." Biochemical and biophysical research communications vol. 492,3 (2017): 528. doi: https://doi.org/10.1016/j.bbrc.2017.08.098
Huang SB, Zhao HD, Wang LF, Sun MF, Zhu YL, Wu YB, Xu YD, Peng SX, Cui C, Shen YQ. Corrigendum to "Intradiencephalon injection of histamine inhibited the recovery of locomotor function of spinal cord injured zebrafish" [Biochem. Biophys. Res. Comm. 489 (1) (2017) 275-280]. Biochem Biophys Res Commun. 2017 Oct 21;492(3):528. doi: 10.1016/j.bbrc.2017.08.098. Epub 2017 Aug 31. PMID: 28864060.
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Corrigendum to "SPOP promotes SIRT2 degradation and suppresses non-small cell lung cancer cell growth" [Biochem. Biophys. Res. Commun. 483 (2017) 880-884].

Biochemical and biophysical research communications

Luo J, Bao YC, Ji XX, Chen B, Deng QF, Zhou SW.
PMID: 28292429
Biochem Biophys Res Commun. 2017 Apr 22;486(1):57. doi: 10.1016/j.bbrc.2017.02.122. Epub 2017 Mar 11.

No abstract available.

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Luo J, Bao YC, Ji XX, et al. Corrigendum to "SPOP promotes SIRT2 degradation and suppresses non-small cell lung cancer cell growth" [Biochem. Biophys. Res. Commun. 483 (2017) 880-884]. Biochem Biophys Res Commun. 2017;486(1):57doi: 10.1016/j.bbrc.2017.02.122.
Luo, J., Bao, Y. C., Ji, X. X., Chen, B., Deng, Q. F., & Zhou, S. W. (2017). Corrigendum to "SPOP promotes SIRT2 degradation and suppresses non-small cell lung cancer cell growth" [Biochem. Biophys. Res. Commun. 483 (2017) 880-884]. Biochemical and biophysical research communications, 486(1), 57. https://doi.org/10.1016/j.bbrc.2017.02.122
Luo, Jie, et al. "Corrigendum to "SPOP promotes SIRT2 degradation and suppresses non-small cell lung cancer cell growth" [Biochem. Biophys. Res. Commun. 483 (2017) 880-884]." Biochemical and biophysical research communications vol. 486,1 (2017): 57. doi: https://doi.org/10.1016/j.bbrc.2017.02.122
Luo J, Bao YC, Ji XX, Chen B, Deng QF, Zhou SW. Corrigendum to "SPOP promotes SIRT2 degradation and suppresses non-small cell lung cancer cell growth" [Biochem. Biophys. Res. Commun. 483 (2017) 880-884]. Biochem Biophys Res Commun. 2017 Apr 22;486(1):57. doi: 10.1016/j.bbrc.2017.02.122. Epub 2017 Mar 11. PMID: 28292429.
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WITHDRAWN: Chronic intermittent hypobaric hypoxia attenuates radiation-induced heart damage in rats.

Biochemical and biophysical research communications

Zhang N, He L, Wang J, Guo Y, Liu Y, Kong Y, Li Y.
PMID: 27372426
Biochem Biophys Res Commun. 2016 Jun 29; doi: 10.1016/j.bbrc.2016.06.145. Epub 2016 Jun 29.

No abstract available.

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Zhang N, He L, Wang J, et al. WITHDRAWN: Chronic intermittent hypobaric hypoxia attenuates radiation-induced heart damage in rats. Biochem Biophys Res Commun. 2016;doi: 10.1016/j.bbrc.2016.06.145.
Zhang, N., He, L., Wang, J., Guo, Y., Liu, Y., Kong, Y., & Li, Y. (2016). WITHDRAWN: Chronic intermittent hypobaric hypoxia attenuates radiation-induced heart damage in rats. Biochemical and biophysical research communications, . https://doi.org/10.1016/j.bbrc.2016.06.145
Zhang, Ning, et al. "WITHDRAWN: Chronic intermittent hypobaric hypoxia attenuates radiation-induced heart damage in rats." Biochemical and biophysical research communications vol. (2016). doi: https://doi.org/10.1016/j.bbrc.2016.06.145
Zhang N, He L, Wang J, Guo Y, Liu Y, Kong Y, Li Y. WITHDRAWN: Chronic intermittent hypobaric hypoxia attenuates radiation-induced heart damage in rats. Biochem Biophys Res Commun. 2016 Jun 29; doi: 10.1016/j.bbrc.2016.06.145. Epub 2016 Jun 29. PMID: 27372426.
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Corrigendum to "Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client" [Biochem. Biophys. Res. Commun. 487 (2) (2017) 375-380].

Biochemical and biophysical research communications

Stan RC, Silva RL, de Camargo MM.
PMID: 28601239
Biochem Biophys Res Commun. 2017 Aug 12;490(1):69. doi: 10.1016/j.bbrc.2017.06.004. Epub 2017 Jun 07.

No abstract available.

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Stan RC, Silva RL, de Camargo MM. Corrigendum to "Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client" [Biochem. Biophys. Res. Commun. 487 (2) (2017) 375-380]. Biochem Biophys Res Commun. 2017;490(1):69doi: 10.1016/j.bbrc.2017.06.004.
Stan, R. C., Silva, R. L., & de Camargo, M. M. (2017). Corrigendum to "Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client" [Biochem. Biophys. Res. Commun. 487 (2) (2017) 375-380]. Biochemical and biophysical research communications, 490(1), 69. https://doi.org/10.1016/j.bbrc.2017.06.004
Stan, Razvan C, et al. "Corrigendum to "Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client" [Biochem. Biophys. Res. Commun. 487 (2) (2017) 375-380]." Biochemical and biophysical research communications vol. 490,1 (2017): 69. doi: https://doi.org/10.1016/j.bbrc.2017.06.004
Stan RC, Silva RL, de Camargo MM. Corrigendum to "Human GRP78 affinity towards its signaling partners Ire1α and PERK is differently modulated by an unfolded protein client" [Biochem. Biophys. Res. Commun. 487 (2) (2017) 375-380]. Biochem Biophys Res Commun. 2017 Aug 12;490(1):69. doi: 10.1016/j.bbrc.2017.06.004. Epub 2017 Jun 07. PMID: 28601239.
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Retraction notice to "UHRF2, another E3 ubiquitin ligase for p53" [Biochem. Biophys. Res. Commun. 425 (2012) 908-911].

Biochemical and biophysical research communications

[No authors listed]
PMID: 24344354
Biochem Biophys Res Commun. 2013 Nov 01;440(4):803. doi: 10.1016/j.bbrc.2013.10.069.

No abstract available.

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Retraction notice to "UHRF2, another E3 ubiquitin ligase for p53" [Biochem. Biophys. Res. Commun. 425 (2012) 908-911]. Biochem Biophys Res Commun. 2013;440(4):803doi: 10.1016/j.bbrc.2013.10.069.
(2013). Retraction notice to "UHRF2, another E3 ubiquitin ligase for p53" [Biochem. Biophys. Res. Commun. 425 (2012) 908-911]. Biochemical and biophysical research communications, 440(4), 803. https://doi.org/10.1016/j.bbrc.2013.10.069
"Retraction notice to "UHRF2, another E3 ubiquitin ligase for p53" [Biochem. Biophys. Res. Commun. 425 (2012) 908-911]." Biochemical and biophysical research communications vol. 440,4 (2013): 803. doi: https://doi.org/10.1016/j.bbrc.2013.10.069
Retraction notice to "UHRF2, another E3 ubiquitin ligase for p53" [Biochem. Biophys. Res. Commun. 425 (2012) 908-911]. Biochem Biophys Res Commun. 2013 Nov 01;440(4):803. doi: 10.1016/j.bbrc.2013.10.069. PMID: 24344354.
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Retracted: Sphingosine kinase-1/sphingosine-1-phosphate receptor type 1 signalling axis is induced by transforming growth factor-β1 and stimulates cell migration in RAW264.7 [Biochem. Biophys. Res. Commun. 426 (2012) 415-420].

Biochemical and biophysical research communications

[No authors listed]
PMID: 24344355
Biochem Biophys Res Commun. 2013 Nov 01;440(4):804. doi: 10.1016/j.bbrc.2013.10.070.

No abstract available.

Cite
Retracted: Sphingosine kinase-1/sphingosine-1-phosphate receptor type 1 signalling axis is induced by transforming growth factor-β1 and stimulates cell migration in RAW264.7 [Biochem. Biophys. Res. Commun. 426 (2012) 415-420]. Biochem Biophys Res Commun. 2013;440(4):804doi: 10.1016/j.bbrc.2013.10.070.
(2013). Retracted: Sphingosine kinase-1/sphingosine-1-phosphate receptor type 1 signalling axis is induced by transforming growth factor-β1 and stimulates cell migration in RAW264.7 [Biochem. Biophys. Res. Commun. 426 (2012) 415-420]. Biochemical and biophysical research communications, 440(4), 804. https://doi.org/10.1016/j.bbrc.2013.10.070
"Retracted: Sphingosine kinase-1/sphingosine-1-phosphate receptor type 1 signalling axis is induced by transforming growth factor-β1 and stimulates cell migration in RAW264.7 [Biochem. Biophys. Res. Commun. 426 (2012) 415-420]." Biochemical and biophysical research communications vol. 440,4 (2013): 804. doi: https://doi.org/10.1016/j.bbrc.2013.10.070
Retracted: Sphingosine kinase-1/sphingosine-1-phosphate receptor type 1 signalling axis is induced by transforming growth factor-β1 and stimulates cell migration in RAW264.7 [Biochem. Biophys. Res. Commun. 426 (2012) 415-420]. Biochem Biophys Res Commun. 2013 Nov 01;440(4):804. doi: 10.1016/j.bbrc.2013.10.070. PMID: 24344355.
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Retraction notice to RETRACTED: NKR-P1A protein, an activating receptor of rat natural killer cells, binds to the chitobiose core of uncompletely glycosylated N-linked glycans, and to linear chitooligomers [Biochem. Biophys. Res. Commun. 238 (1997) 149–153].

Biochemical and biophysical research communications

[No authors listed]
PMID: 25658013
Biochem Biophys Res Commun. 2014 Oct 24;453(3):680. doi: 10.1016/j.bbrc.2014.08.021.

No abstract available.

Cite
Retraction notice to RETRACTED: NKR-P1A protein, an activating receptor of rat natural killer cells, binds to the chitobiose core of uncompletely glycosylated N-linked glycans, and to linear chitooligomers [Biochem. Biophys. Res. Commun. 238 (1997) 149–153]. Biochem Biophys Res Commun. 2014;453(3):680doi: 10.1016/j.bbrc.2014.08.021.
(2014). Retraction notice to RETRACTED: NKR-P1A protein, an activating receptor of rat natural killer cells, binds to the chitobiose core of uncompletely glycosylated N-linked glycans, and to linear chitooligomers [Biochem. Biophys. Res. Commun. 238 (1997) 149–153]. Biochemical and biophysical research communications, 453(3), 680. https://doi.org/10.1016/j.bbrc.2014.08.021
"Retraction notice to RETRACTED: NKR-P1A protein, an activating receptor of rat natural killer cells, binds to the chitobiose core of uncompletely glycosylated N-linked glycans, and to linear chitooligomers [Biochem. Biophys. Res. Commun. 238 (1997) 149–153]." Biochemical and biophysical research communications vol. 453,3 (2014): 680. doi: https://doi.org/10.1016/j.bbrc.2014.08.021
Retraction notice to RETRACTED: NKR-P1A protein, an activating receptor of rat natural killer cells, binds to the chitobiose core of uncompletely glycosylated N-linked glycans, and to linear chitooligomers [Biochem. Biophys. Res. Commun. 238 (1997) 149–153]. Biochem Biophys Res Commun. 2014 Oct 24;453(3):680. doi: 10.1016/j.bbrc.2014.08.021. PMID: 25658013.
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WITHDRAWN: Salvianolate attenuates renal fibrosis in rat models of diabetic nephropathy by inhibiting inflammation and oxidative stress mechanisms.

Biochemical and biophysical research communications

Xiong C, Lu J, Wang X, Masucci MV.
PMID: 28435064
Biochem Biophys Res Commun. 2017 Apr 20; doi: 10.1016/j.bbrc.2017.04.101. Epub 2017 Apr 20.

This article has been withdrawn at the request of the author(s) and/or editor. The Publisher apologizes for any inconvenience this may cause. The full Elsevier Policy on Article Withdrawal can be found at https://www.elsevier.com/about/our-business/policies/article-withdrawal.

Cite
Xiong C, Lu J, Wang X, et al. WITHDRAWN: Salvianolate attenuates renal fibrosis in rat models of diabetic nephropathy by inhibiting inflammation and oxidative stress mechanisms. Biochem Biophys Res Commun. 2017;doi: 10.1016/j.bbrc.2017.04.101.
Xiong, C., Lu, J., Wang, X., & Masucci, M. V. (2017). WITHDRAWN: Salvianolate attenuates renal fibrosis in rat models of diabetic nephropathy by inhibiting inflammation and oxidative stress mechanisms. Biochemical and biophysical research communications, . https://doi.org/10.1016/j.bbrc.2017.04.101
Xiong, Chongxiang, et al. "WITHDRAWN: Salvianolate attenuates renal fibrosis in rat models of diabetic nephropathy by inhibiting inflammation and oxidative stress mechanisms." Biochemical and biophysical research communications vol. (2017). doi: https://doi.org/10.1016/j.bbrc.2017.04.101
Xiong C, Lu J, Wang X, Masucci MV. WITHDRAWN: Salvianolate attenuates renal fibrosis in rat models of diabetic nephropathy by inhibiting inflammation and oxidative stress mechanisms. Biochem Biophys Res Commun. 2017 Apr 20; doi: 10.1016/j.bbrc.2017.04.101. Epub 2017 Apr 20. PMID: 28435064.
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Sevoflurane pretreatment attenuates TNF-α-induced human endothelial cell dysfunction through activating eNOS/NO pathway.

Biochemical and biophysical research communications

Li S, Xu J, Yao W, Li H, Liu Q, Xiao F, Irwin MG, Xia Z, Ruan W.
PMID: 25838201
Biochem Biophys Res Commun. 2015 May 08;460(3):879-86. doi: 10.1016/j.bbrc.2015.03.126. Epub 2015 Mar 31.

Endothelial dysfunction induced by oxidative stress and inflammation plays a critical role in the pathogenesis of cardiovascular diseases. The anesthetic sevoflurane confers cytoprotective effects through its anti-inflammatory properties in various pathologies such as systemic inflammatory response syndrome and ischemic-reperfusion...

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Li S, Xu J, Yao W, et al. Sevoflurane pretreatment attenuates TNF-α-induced human endothelial cell dysfunction through activating eNOS/NO pathway. Biochem Biophys Res Commun. 2015;460(3):879-86doi: 10.1016/j.bbrc.2015.03.126.
Li, S., Xu, J., Yao, W., Li, H., Liu, Q., Xiao, F., Irwin, M. G., Xia, Z., & Ruan, W. (2015). Sevoflurane pretreatment attenuates TNF-α-induced human endothelial cell dysfunction through activating eNOS/NO pathway. Biochemical and biophysical research communications, 460(3), 879-86. https://doi.org/10.1016/j.bbrc.2015.03.126
Li, Suobei, et al. "Sevoflurane pretreatment attenuates TNF-α-induced human endothelial cell dysfunction through activating eNOS/NO pathway." Biochemical and biophysical research communications vol. 460,3 (2015): 879-86. doi: https://doi.org/10.1016/j.bbrc.2015.03.126
Li S, Xu J, Yao W, Li H, Liu Q, Xiao F, Irwin MG, Xia Z, Ruan W. Sevoflurane pretreatment attenuates TNF-α-induced human endothelial cell dysfunction through activating eNOS/NO pathway. Biochem Biophys Res Commun. 2015 May 08;460(3):879-86. doi: 10.1016/j.bbrc.2015.03.126. Epub 2015 Mar 31. PMID: 25838201.
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Transfer RNA Is the Source of Extracellular Isopentenyladenine in a Ti-Plasmidless Strain of Agrobacterium tumefaciens.

Plant physiology

Gray J, Gelvin SB, Meilan R, Morris RO.
PMID: 12226194
Plant Physiol. 1996 Feb;110(2):431-438. doi: 10.1104/pp.110.2.431.

Even in the absence of the classical Ti plasmid-encoded cytokinin biosynthetic genes ipt and tzs, Agrobacterium tumefaciens strains still release significant amounts of the cytokinin isopentenyladenine (iP) into the culture medium (R.W. Kaiss-Chapman and R.O. Morris [1977] Biochem Biophys...

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Gray J, Gelvin SB, Meilan R, et al. Transfer RNA Is the Source of Extracellular Isopentenyladenine in a Ti-Plasmidless Strain of Agrobacterium tumefaciens. Plant Physiol. 1996;110(2):431-438doi: 10.1104/pp.110.2.431.
Gray, J., Gelvin, S. B., Meilan, R., & Morris, R. O. (1996). Transfer RNA Is the Source of Extracellular Isopentenyladenine in a Ti-Plasmidless Strain of Agrobacterium tumefaciens. Plant physiology, 110(2), 431-438. https://doi.org/10.1104/pp.110.2.431
Gray, J., et al. "Transfer RNA Is the Source of Extracellular Isopentenyladenine in a Ti-Plasmidless Strain of Agrobacterium tumefaciens." Plant physiology vol. 110,2 (1996): 431-438. doi: https://doi.org/10.1104/pp.110.2.431
Gray J, Gelvin SB, Meilan R, Morris RO. Transfer RNA Is the Source of Extracellular Isopentenyladenine in a Ti-Plasmidless Strain of Agrobacterium tumefaciens. Plant Physiol. 1996 Feb;110(2):431-438. doi: 10.1104/pp.110.2.431. PMID: 12226194; PMCID: PMC157737.
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Distribution of neuromedin U receptor subtype 2 mRNA in the rat brain.

Brain research. Gene expression patterns

Guan XM, Yu H, Jiang Q, Van Der Ploeg LH, Liu Q.
PMID: 15018811
Brain Res Gene Expr Patterns. 2001 Aug;1(1):1-4. doi: 10.1016/s1567-133x(00)00002-8.

Neuromedin U (NMU) is a family of peptides found in the gut and the central nervous system [Neuroscience 25 (1988) 797; Biochem. Biophys. Res. Commun. 130 (1985) 1078]. While several peripheral activities such as uterus stimulating and hypertensive effects...

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Guan XM, Yu H, Jiang Q, et al. Distribution of neuromedin U receptor subtype 2 mRNA in the rat brain. Brain Res Gene Expr Patterns. 2001;1(1):1-4doi: 10.1016/s1567-133x(00)00002-8.
Guan, X. M., Yu, H., Jiang, Q., Van Der Ploeg, L. H., & Liu, Q. (2001). Distribution of neuromedin U receptor subtype 2 mRNA in the rat brain. Brain research. Gene expression patterns, 1(1), 1-4. https://doi.org/10.1016/s1567-133x(00)00002-8
Guan, X M, et al. "Distribution of neuromedin U receptor subtype 2 mRNA in the rat brain." Brain research. Gene expression patterns vol. 1,1 (2001): 1-4. doi: https://doi.org/10.1016/s1567-133x(00)00002-8
Guan XM, Yu H, Jiang Q, Van Der Ploeg LH, Liu Q. Distribution of neuromedin U receptor subtype 2 mRNA in the rat brain. Brain Res Gene Expr Patterns. 2001 Aug;1(1):1-4. doi: 10.1016/s1567-133x(00)00002-8. PMID: 15018811.
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WITHDRAWN: Expression of p53, p63, and p73 isoforms in squamous cell carcinoma and adenocarcinoma of esophagus☆,☆☆.

Biochemical and biophysical research communications

Cui R, He J, Mei R, de Fromentel CC, Martel-Planche G, Taniere P, Hainaut P.
PMID: 16126170
Biochem Biophys Res Commun. 2005 Aug 22;336(1):339-345. doi: 10.1016/j.bbrc.2005.08.084. Epub 2005 Aug 22.

This article has been withdrawn consistent with Elsevier Policy on Article Withdrawal (http://www.elsevier.com/locate/withdrawalpolicy). The Publisher apologizes for any inconvenience this may cause.

Cite
Cui R, He J, Mei R, et al. WITHDRAWN: Expression of p53, p63, and p73 isoforms in squamous cell carcinoma and adenocarcinoma of esophagus☆,☆☆. Biochem Biophys Res Commun. 2005;336(1):339-345doi: 10.1016/j.bbrc.2005.08.084.
Cui, R., He, J., Mei, R., de Fromentel, C. C., Martel-Planche, G., Taniere, P., & Hainaut, P. (2005). WITHDRAWN: Expression of p53, p63, and p73 isoforms in squamous cell carcinoma and adenocarcinoma of esophagus☆,☆☆. Biochemical and biophysical research communications, 336(1), 339-345. https://doi.org/10.1016/j.bbrc.2005.08.084
Cui, Rutao, et al. "WITHDRAWN: Expression of p53, p63, and p73 isoforms in squamous cell carcinoma and adenocarcinoma of esophagus☆,☆☆." Biochemical and biophysical research communications vol. 336,1 (2005): 339-345. doi: https://doi.org/10.1016/j.bbrc.2005.08.084
Cui R, He J, Mei R, de Fromentel CC, Martel-Planche G, Taniere P, Hainaut P. WITHDRAWN: Expression of p53, p63, and p73 isoforms in squamous cell carcinoma and adenocarcinoma of esophagus☆,☆☆. Biochem Biophys Res Commun. 2005 Aug 22;336(1):339-345. doi: 10.1016/j.bbrc.2005.08.084. Epub 2005 Aug 22. PMID: 16126170.
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Showing 1 to 12 of 1598 entries